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Status Epilepticus in a Patient with Extrapontine Myelinolysis
Àå±â¹®, ¿ì³ªÀº, ÀÌÇýÁ¤, ±è´ÙÀº, À¯ÇöÁö, ÇÑÈñÁ¶, ±è±Ô½Ä,
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Àå±â¹® ( Jang Ki-Moon )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
¿ì³ªÀº ( Woo Na-Eun )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
ÀÌÇýÁ¤ ( Lee Hye-Jeong )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
±è´ÙÀº ( Kim Da-Eun )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
À¯ÇöÁö ( Lyou Hyun-Ji )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
ÇÑÈñÁ¶ ( Han Hee-Jo )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
±è±Ô½Ä ( Kim Gyu-Sik )
±¹¹Î°Ç°º¸Çè Àϻ꺴¿ø ½Å°æ°ú
Abstract
Extrapontine myelinolysis is a demyelinating disorder usually caused by rapid correction of chronic hyponatremia. We present an 82-year-old female patient with general weakness, and severe hyponatremia (103 mEq/L) which was corrected 14 mEq/L during 21 hours. She was stuporous and presented status epilepticus. Brain T2-weighted image showed high signal intensities of bilateral thalami and electroencephalogram monitoring indicated right hemisphere lateralized periodic discharges, maximal in the right frontal region. This case shows that status epilepticus can occur in a patient of extrapontine myelinolysis involved bilateral thalami without pontine lesions.
Å°¿öµå
Myelinolysis; central pontine; Status epilepticus; Hyponatremia
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