Àá½Ã¸¸ ±â´Ù·Á ÁÖ¼¼¿ä. ·ÎµùÁßÀÔ´Ï´Ù.

±Þ¼º ³ú°æ»öÁõ¿¡¼­ Ç×ÀÀ°íÁ¦³ª Ç÷Àü ¿ëÇØÁ¦ »ç¿ëÈÄ ¹ß»ýÇÑ ±Þ¼º ³ú°æ¸·ÇÏÇ÷Á¾ Acute Subdural Hematoma Developed During Anticoagulant of Thrombolytic Therapy in Patients with Cerbral Infarction

´ëÇѽŰæ¿Ü°úÇÐȸÁö 1996³â 25±Ç 1È£ p.184 ~ 187
¼Ò¼Ó »ó¼¼Á¤º¸
±è±Ùâ ÀÓ¿µÁø/±èÇüµÎ/±èżº/±è±¹±â/À̺À¾Ï/ÀÓ¾ð

Abstract


Patients with cardiogenic embolic stroke may experience an early. recurrent cerebral embolism. Fortunately. limited evidence suggests that anticoagulatory agents or thrombolytic agents may prevent recurrent cardiogenic emboli and halt progression
of so
called "progressing stroke"
However. because of the possibility of the intracerebral hemorrhage. use of such agents has generally been considered cautiously with timing. dosage and patient selection.
Serious complications of anticoagulation for presumed embolic stroke are hemorrhage in the area of infarction.
We experlenced two patients with nonseptic cerebral embolism of cardiac origin. They were managed with anticoagulant or thrombolytic therapy. but resulted in clinical deterioration or death from spontaneous subdural hemorrhage.
In each patient. an initial CT scan excluded the presence of hemorrhage but a second CT scan after clinical deterioration. documented subdural hemorrhage.

Å°¿öµå

¿ø¹® ¹× ¸µÅ©¾Æ¿ô Á¤º¸

 

µîÀçÀú³Î Á¤º¸

KoreaMed
KAMS