Àá½Ã¸¸ ±â´Ù·Á ÁÖ¼¼¿ä. ·ÎµùÁßÀÔ´Ï´Ù.

Obesity, Inflammation and Diet

´ëÇѼҾƼÒÈ­±â¿µ¾çÇÐȸÁö 2013³â 16±Ç 3È£ p.143 ~ 152
ÀÌÇѼÛÀÌ, Á¶¿©¿ø, ÀÌÀμ®,
¼Ò¼Ó »ó¼¼Á¤º¸
ÀÌÇѼÛÀÌ ( Lee Hansongyi ) 
Kyung Hee University Graduate School of East-West Medical Science Department of Medical Nutrition

Á¶¿©¿ø ( Choue Ryo-Won ) 
Kyung Hee University Graduate School of East-West Medical Science Department of Medical Nutrition
ÀÌÀμ® ( Lee In-Seok ) 
Kyung Hee University Medical Center Nutrition Team

Abstract


Obesity is a state in which there is an over-accumulation of subcutaneous and/or abdominal adipose tissue. This adipose tissue is no longer considered inert and mainly devoted to storing energy; it is emerging as an active tissue in the regulation of physiological and pathological processes, including immunity and inflammation. Adipose tissue produces and releases a variety of adipokines (leptin, adiponectin, resistin, and visfatin), as well as pro- and anti-in-flammatory cytokines (tumor necrosis factor-?, interleukin [IL]-4, IL-6, and others). Adipose tissue is also implicated in the development of chronic metabolic diseases such as type 2 diabetes mellitus or cardiovascular disease. Obesity is thus an underlying condition for inflammatory and metabolic diseases. Diet or dietary patterns play critical roles in obesity and other pathophysiological conditions. A healthy diet and some nutrients are generally considered benefi-cial; however, some dietary nutrients are still considered controversial. In this article, dietary factors that influence inflammation associated with obesity are discussed.

Å°¿öµå

Obesity;Adipose tissue;Inflammation;Diet;Epidemiology;Clinical trial

¿ø¹® ¹× ¸µÅ©¾Æ¿ô Á¤º¸

  

µîÀçÀú³Î Á¤º¸

KCI
KoreaMed
KAMS