Obesity, Inflammation and Diet
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ÀÌÇѼÛÀÌ ( Lee Hansongyi )
Kyung Hee University Graduate School of East-West Medical Science Department of Medical Nutrition
Á¶¿©¿ø ( Choue Ryo-Won )
Kyung Hee University Graduate School of East-West Medical Science Department of Medical Nutrition
ÀÌÀμ® ( Lee In-Seok )
Kyung Hee University Medical Center Nutrition Team
KMID : 0816120130160030143
Abstract
Obesity is a state in which there is an over-accumulation of subcutaneous and/or abdominal adipose tissue. This adipose tissue is no longer considered inert and mainly devoted to storing energy; it is emerging as an active tissue in the regulation of physiological and pathological processes, including immunity and inflammation. Adipose tissue produces and releases a variety of adipokines (leptin, adiponectin, resistin, and visfatin), as well as pro- and anti-in-flammatory cytokines (tumor necrosis factor-?, interleukin [IL]-4, IL-6, and others). Adipose tissue is also implicated in the development of chronic metabolic diseases such as type 2 diabetes mellitus or cardiovascular disease. Obesity is thus an underlying condition for inflammatory and metabolic diseases. Diet or dietary patterns play critical roles in obesity and other pathophysiological conditions. A healthy diet and some nutrients are generally considered benefi-cial; however, some dietary nutrients are still considered controversial. In this article, dietary factors that influence inflammation associated with obesity are discussed.
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Obesity;Adipose tissue;Inflammation;Diet;Epidemiology;Clinical trial
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