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Àå±â°£ ´çÁ¶Àý Á¤µµ°¡ ´ç´¢º´¼º ½Å°æº´ÁõÀÇ ½Å°æÀüµµ¿¡ ¹ÌÄ¡´Â ¿µÇâ The Effect of Glucose Long-Term Control on the Nerve Conduction Parameters in the Diabetic Peripheral Neuropathy

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¹ÚÀº¹Ì ( Park Eun-Mi ) 
°í·Á´ëÇб³ ÀÇ°ú´ëÇÐ ÀçÈ°ÀÇÇб³½Ç

±è¼¼ÁÖ ( Kim Sei-Joo ) 
°í·Á´ëÇб³ ÀÇ°ú´ëÇÐ ÀçÈ°ÀÇÇб³½Ç
¼­°ü½Ä ( Seo Gwan-Sik ) 
°í·Á´ëÇб³ ÀÇ°ú´ëÇÐ ÀçÈ°ÀÇÇб³½Ç
½É±ÔÈÆ ( Sim Kyu-Hun ) 
°í·Á´ëÇб³ ÀÇ°ú´ëÇÐ ÀçÈ°ÀÇÇб³½Ç
À±ÁؽĠ( Yoon Joon-Shik ) 
°í·Á´ëÇб³ ÀÇ°ú´ëÇÐ ÀçÈ°ÀÇÇб³½Ç

Abstract


Objectives: In order to assess the correlation between the glucose control and the change of nerve conduction
in the diabetic peripheral neuropathy.

Method: Thirty five patients with diabetic peripheral neuropathy diagnosed by nerve conduction study
(NCS) were included and NCS were reexamined later. The values of nerve conduction were evaluated by
difference between 1st study and follow-up study. The level of fasting blood glucose is divided to 10
grades and level of HbA1c is divided to 6 grades according to goal of glucose control by American diabetes
association. The effect of glucose control on the change of nerve conduction were analyzed by linear
regression test.

Results: The controlled level of fasting blood glucose had a significant effect on the latencies of median,
ulnar motor and sensory nerves and sural sensory nerve. The level of HbA1c had a significant effect on
the latency of peroneal motor nerve (p<0.05).

Conclusion: The level of fasting blood glucose and HbA1c had a significant effect on the change of
nerve conduction latency.

Å°¿öµå

Glucose Control;Diabetic Peripheral Neuropathy;Nerve Conduction Study;Correlation

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